Anti-Ter-119 MicroBeads are suitable for positive selection or depletion of mouse erythrocytes or erythroid progenitors from lymphoid tissues.

Data and images for Anti-Ter-119 MicroBeads, mouse

Figures

Figure 1

Isolation of erythrocytes from a mouse spleen cell suspension using Anti-Ter-119 MicroBeads, a MiniMACS™ Separator, and an MS Column.
Spleen cells before separation
Isolated Ter-119
+
erythrocytes
View details

Figure 1

Isolation of erythrocytes from a mouse spleen cell suspension using Anti-Ter-119 MicroBeads, a MiniMACS™ Separator, and an MS Column.
View details

Figure 1

Isolation of erythrocytes from a mouse spleen cell suspension using Anti-Ter-119 MicroBeads, a MiniMACS™ Separator, and an MS Column.

Specifications for Anti-Ter-119 MicroBeads, mouse

Overview

Anti-Ter-119 MicroBeads are suitable for positive selection or depletion of mouse erythrocytes or erythroid progenitors from lymphoid tissues.

Detailed product information

Applications

Anti-Ter-119 MicroBeads were used for positive selection
1,4
or depletion
2,3
of mature erythrocytes and erythroid precursor cells from mouse bone marrow, fetal liver, or spleen. They were also used in combination with CD45 MicroBeads for enrichment of human cells from bone marrow of chimeric mice by depletion of mouse hematopoietic cells
1
, for isolation of erythrocytes from immunodeficient mice
2
, and for depletion of red blood cells as an alternative method to osmotic lysis
3
. Furthermore, they were used to isolate erythrocytes in an experimental mouse model of malaria.
4

Columns

For positive selection: MS, LS, XS, or autoMACS
®
Columns. For depletion: LD, D, or autoMACS Columns.

References for Anti-Ter-119 MicroBeads, mouse

Publications

  1. Schiedlmeier, B. et al. (2000) Quantitative assessment of retroviral transfer of the human multidrug resistance 1 gene to human mobilized peripheral blood progenitor cells engrafted in nonobese diabetic/severe combined immunodeficient mice. Blood 95: 1237-1248
  2. Chan, J. Y. et al. (2001) Reduced oxidative-stress response in red blood cells from p45NFE2-deficient mice. Blood 97: 2151-2158
  3. Ye, S. K. et al. (1999) Induction of germline transcription in the TCRgamma locus by Stat5: implications for accessibility control by the IL-7 receptor. Immunity 11: 213-223
  4. Chang, K. H. et al. (2004) Inappropriately low reticulocytosis in severe malarial anemia correlates with suppression in the development of late erythroid precursors. Blood 103: 3727-3735

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